VACCINE VIOLENCE: THE VACCINES AND AUTISM STORY
vaccines and autism, vaccines and neurodevelopmental disorders, vaccine composition and chemicals, vaccine related baby deaths - updated 31 May 2016
 
 
Multiple Vaccine Schedule Never Tested - Your Child is the Test
Multiple vaccines schedule never tested
 
 
It's More Than Just MMR: How Current Practices Destroy the Health of Our Children
synergistic effects of vaccines and medical interventions on neurodevelopmental health
 
 
Andrew Wakefield's 1998 Case Study
1998 Case Study Not the First Time Vaccines and Autism Questioned
 
 
Dr. Andrew Wakefield's Further Research
Follow up Research done by Dr. Andrew Wakefield
 
 
Doctor Andrew Wakefield Addresses Allegations Against Him
Updated 14 August 2016.
 
 
Other Research into Autism, Bowel Disease, Auto-Immunity and Viral Antigens
 
 
Immuno-Suppression of Measles Virus and its Connection to Autism
 
 
Yes, Thimerosal CAN be a Cause of Autism
Studies showing thimerosal/autism link
 
 
It's the Aluminium Too
Aluminium and Thimerosal Combined Have NEVER been Studied
 
 
The Fetal Tissue and MMR Link to Autism
Human fetal tissue, MMR and autoantibodies in Autism
 
 
Vaccines, Chronic Brain Activation and Autism
Vaccines trigger the brain's immune system
 
 
Gender-Specific Issues in Autism and Vaccination
Why do so many boys get autism?
 
 
'The Benefits Outweigh the Risk' - But They Don't Know What the Risks Are
Did bacteriophages and polyoma viruses in vaccines contribute to autism?
 
 
Autism and TH1/TH2 Balance
Updated 14 August 2016
 
 
VAXXED - The film about the CDC Coverup of MMR and Autism that the Government Didn't Want You to See
 
 
Watch Vaxxed The Movie
 
 
Hear the Silence - 2003 Film
Dramatised Account of MMR/Autism Story that was Later Banned from TV
 
 
Speech by Robert F Kennedy Junior about CDC Corruption
New page 6 September 2016
 
 
Mitochondrial Disorder Isn't Just Genetic - Vaccines Alter DNA and Can Cause the Dysfunction
Short Documentary
 
 
Triplets All Become Autistic on the Same Day Within Hours of Prevenar Vaccination
 
 

It's the Aluminium Too

According to Neurochemical Research, the adjuvant aluminium has NEVER been studied when combined with thimerosal, something that happens with vaccination even when it is in trace amounts:

'The neurotoxic effect of ethylmercury has not been studied with co-occurring adjuvant-Al in TCVs; (c) animal studies have shown that exposure to Thimerosal-Hg can lead to accumulation of inorganic Hg in brain, and that (d) doses relevant to TCV exposure possess the potential to affect human neuro-development. Thimerosal at concentrations relevant for infants' exposure (in vaccines) is toxic to cultured human-brain cells and to laboratory animals.'

Considering that they inject trace amounts of thimerosal and aluminium into babies on a mass scale you would have thought they would have bothered to do the tests BEFORE they combined mercury with aluminium and told the world's parents it was safe to inject into their children repeatedly.

In fact, thimerosal as a stand alone product in vaccines was only tested once in the 1920's before a multiple vaccine schedule containing thimerosal mixed with aluminium was introduced. The synergistic effects of mercury and aluminium are only just beginning to receive attention from the medical community.

http://www.safeminds.org/protect-yourself/fludocuments/ACIP%20Letter%20AJN%20edits%207-06.pdf

Aluminium and Mercury Combined Cause an Increase in Cell Death

Dr. Haley studied the synergistic effects of mercury and aluminium combined. He wrote:

'We therefore investigated the possible involvement of aluminum cation (found in vaccines), antibiotics (neomycin) and male versus female (estrogen versus testosterone) on the toxic effects of 50 nanomolar (nM) thimerosal on neurons in culture. Neurons can be cultured for 24 hours without much death (Fig. 6). Fifty nanomolar thimerosal alone (solid circles[●]) will cause the death of about 70% of the neurons within 24 hours. The synergistic effects of aluminum, neomycin and testosterone are shown (Fig.6) and are as follows:

Aluminum: Aluminum hydroxide alone (solid triangles []) at 500 nM showed no significant death of cells at 6 hours, and only slight toxicity over the 24-hour period. Thimerosal at 50 nM effected only a slight increase e in neuron death at 6 hours.

However, in the presence of 50 nM thimerosal plus 500 nM aluminum hydroxide (open triangles [Δ]), the neuronal death increases toroughly 60%, an amazing increase and clearly demonstrates the synergistic effects of other metals on mercury toxicity and certainly thimerosal toxicity.'

Source:

http://homeoint.ru/pdfs/haley.pdf

In a paper entitled 'Aluminium Induced Entropy in Biological Systems', the authors point out:

‘Aluminium (Al3+), suspected as a toxicant for 100 years, injures the CNS and immune systems, individually and synergistically…Al3+ disrupts biological water dynamics and macromolecules: DNAs, RNAs, proteoglycans and proteins. Al3+ disrupts H-bond cooperativity interfering with the quantum coherence of living systems. Al3+ interferes with biological signaling – biosemiosis – from the very lowest to the highest levels in the nervous system. The effects are synergistic with other toxicants, including mercury, lead, fluoride, and glyphosate.’

Source:

http://scholar.google.co.uk/scholar?start=10&q=synergistic+effect+of+aluminium+and+thimerosal&hl=en&as_sdt=0,5&as_vis=1

So the aluminium in vaccines actually makes the trace amounts of thimerosal even more dangerous!

Mechanisms of aluminum adjuvant toxicity and autoimmunity in pediatric populations

Immune challenges during early development, including those vaccine-induced, can lead to permanent detrimental alterations of the brain and immune function. Experimental evidence also shows that simultaneous administration of as little as two to three immune adjuvants can overcome genetic resistance to autoimmunity. In some developed countries, by the time children are 4 to 6 years old, they will have received a total of 126 antigenic compounds along with high amounts of aluminum (Al) adjuvants through routine vaccinations. According to the US Food and Drug Administration, safety assessments for vaccines have often not included appropriate toxicity studies because vaccines have not been viewed as inherently toxic. Taken together, these observations raise plausible concerns about the overall safety of current childhood vaccination programs. When assessing adjuvant toxicity in children, several key points ought to be considered: (i) infants and children should not be viewed as "small adults" with regard to toxicological risk as their unique physiology makes them much more vulnerable to toxic insults; (ii) in adult humans Al vaccine adjuvants have been linked to a variety of serious autoimmune and inflammatory conditions (i.e., "ASIA"), yet children are regularly exposed to much higher amounts of Al from vaccines than adults; (iii) it is often assumed that peripheral immune responses do not affect brain function. However, it is now clearly established that there is a bidirectional neuro-immune cross-talk that plays crucial roles in immunoregulation as well as brain function. In turn, perturbations of the neuro-immune axis have been demonstrated in many autoimmune diseases encompassed in "ASIA" and are thought to be driven by a hyperactive immune response; and (iv) the same components of the neuro-immune axis that play key roles in brain development and immune function are heavily targeted by Al adjuvants. In summary, research evidence shows that increasing concerns about current vaccination practices may indeed be warranted. Because children may be most at risk of vaccine-induced complications, a rigorous evaluation of the vaccine-related adverse health impacts in the pediatric population is urgently needed.

Source: Lupus. 2012;21(2):223-30

http://www.ncbi.nlm.nih.gov/pubmed/22235057

Aluminum vaccine adjuvants: are they safe?

Aluminum is an experimentally demonstrated neurotoxin and the most commonly used vaccine adjuvant. Despite almost 90 years of widespread use of aluminum adjuvants, medical science's understanding about their mechanisms of action is still remarkably poor. There is also a concerning scarcity of data on toxicology and pharmacokinetics of these compounds. In spite of this, the notion that aluminum in vaccines is safe appears to be widely accepted. Experimental research, however, clearly shows that aluminum adjuvants have a potential to induce serious immunological disorders in humans. In particular, aluminum in adjuvant form carries a risk for autoimmunity, long-term brain inflammation and associated neurological complications and may thus have profound and widespread adverse health consequences. In our opinion, the possibility that vaccine benefits may have been overrated and the risk of potential adverse effects underestimated, has not been rigorously evaluated in the medical and scientific community. We hope that the present paper will provide a framework for a much needed and long overdue assessment of this highly contentious medical issue.

Source: Curr Med Chem. 2011;18(17):2630-7

http://www.ncbi.nlm.nih.gov/pubmed/21568886

Aluminum in the central nervous system (CNS): toxicity in humans and animals, vaccine adjuvants, and autoimmunity.

Abstract

We have examined the neurotoxicity of aluminum in humans and animals under various conditions, following different routes of administration, and provide an overview of the various associated disease states. The literature demonstrates clearly negative impacts of aluminum on the nervous system across the age span. In adults, aluminum exposure can lead to apparently age-related neurological deficits resembling Alzheimer's and has been linked to this disease and to the Guamanian variant, ALS-PDC. Similar outcomes have been found in animal models. In addition, injection of aluminum adjuvants in an attempt to model Gulf War syndrome and associated neurological deficits leads to an ALS phenotype in young male mice. In young children, a highly significant correlation exists between the number of pediatric aluminum-adjuvanted vaccines administered and the rate of autism spectrum disorders. Many of the features of aluminum-induced neurotoxicity may arise, in part, from autoimmune reactions, as part of the ASIA syndrome.

Source: Immunol Res. 2013 Jul;56(2-3):304-16. doi: 10.1007/s12026-013-8403-1.

http://www.ncbi.nlm.nih.gov/pubmed/23609067

Aluminum adjuvant linked to Gulf War illness induces motor neuron death in mice.

Abstract

Gulf War illness (GWI) affects a significant percentage of veterans of the 1991 conflict, but its origin remains unknown. Associated with some cases of GWI are increased incidences of amyotrophic lateral sclerosis and other neurological disorders. Whereas many environmental factors have been linked to GWI, the role of the anthrax vaccine has come under increasing scrutiny. Among the vaccine's potentially toxic components are the adjuvants aluminum hydroxide and squalene. To examine whether these compounds might contribute to neuronal deficits associated with GWI, an animal model for examining the potential neurological impact of aluminum hydroxide, squalene, or aluminum hydroxide combined with squalene was developed. Young, male colony CD-1 mice were injected with the adjuvants at doses equivalent to those given to US military service personnel. All mice were subjected to a battery of motor and cognitive-behavioral tests over a 6-mo period postinjections. Following sacrifice, central nervous system tissues were examined using immunohistochemistry for evidence of inflammation and cell death. Behavioral testing showed motor deficits in the aluminum treatment group that expressed as a progressive decrease in strength measured by the wire-mesh hang test (final deficit at 24 wk; about 50%). Significant cognitive deficits in water-maze learning were observed in the combined aluminum and squalene group (4.3 errors per trial) compared with the controls (0.2 errors per trial) after 20 wk. Apoptotic neurons were identified in aluminum-injected animals that showed significantly increased activated caspase-3 labeling in lumbar spinal cord (255%) and primary motor cortex (192%) compared with the controls. Aluminum-treated groups also showed significant motor neuron loss (35%) and increased numbers of astrocytes (350%) in the lumbar spinal cord. The findings suggest a possible role for the aluminum adjuvant in some neurological features associated with GWI and possibly an additional role for the combination of adjuvants.

Source: Neuromolecular Med. 2007;9(1):83-100.

http://www.ncbi.nlm.nih.gov/pubmed/17114826

Autoimmune/autoinflammatory syndrome induced by adjuvants (ASIA syndrome) in commercial sheep.

Abstract

We describe a form of the autoimmune/autoinflammatory syndrome induced by adjuvants (ASIA syndrome) in commercial sheep, linked to the repetitive inoculation of aluminum-containing adjuvants through vaccination. The syndrome shows an acute phase that affects less than 0.5% of animals in a given herd, it appears 2-6 days after an adjuvant-containing inoculation and it is characterized by an acute neurological episode with low response to external stimuli and acute meningoencephalitis, most animals apparently recovering afterward. The chronic phase is seen in a higher proportion of flocks, it can follow the acute phase, and it is triggered by external stimuli, mostly low temperatures. The chronic phase begins with an excitatory phase, followed by weakness, extreme cachexia, tetraplegia and death. Gross lesions are related to a cachectic process with muscular atrophy, and microscopic lesions are mostly linked to a neurodegenerative process in both dorsal and ventral column of the gray matter of the spinal cord. Experimental reproduction of ovine ASIA in a small group of repeatedly vaccinated animals was successful. Detection of Al(III) in tissues indicated the presence of aluminum in the nervous tissue of experimental animals. The present report is the first description of a new sheep syndrome (ovine ASIA syndrome) linked to multiple, repetitive vaccination and that can have devastating consequences as it happened after the compulsory vaccination against bluetongue in 2008. The ovine ASIA syndrome can be used as a model of other similar diseases affecting both human and animals. A major research effort is needed in order to understand its complex pathogenesis.

Source: Immunol Res. 2013 Jul;56(2-3):317-24. doi: 10.1007/s12026-013-8404-0

http://www.ncbi.nlm.nih.gov/pubmed/23579772

Aluminum hydroxide injections lead to motor deficits and motor neuron degeneration.

Abstract

Gulf War Syndrome is a multi-system disorder afflicting many veterans of Western armies in the 1990-1991 Gulf War. A number of those afflicted may show neurological deficits including various cognitive dysfunctions and motor neuron disease, the latter expression virtually indistinguishable from classical amyotrophic lateral sclerosis (ALS) except for the age of onset. This ALS "cluster" represents the second such ALS cluster described in the literature to date. Possible causes of GWS include several of the adjuvants in the anthrax vaccine and others. The most likely culprit appears to be aluminum hydroxide. In an initial series of experiments, we examined the potential toxicity of aluminum hydroxide in male, outbred CD-1 mice injected subcutaneously in two equivalent-to-human doses. After sacrifice, spinal cord and motor cortex samples were examined by immunohistochemistry. Aluminum-treated mice showed significantly increased apoptosis of motor neurons and increases in reactive astrocytes and microglial proliferation within the spinal cord and cortex. Morin stain detected the presence of aluminum in the cytoplasm of motor neurons with some neurons also testing positive for the presence of hyper-phosphorylated tau protein, a pathological hallmark of various neurological diseases, including Alzheimer's disease and frontotemporal dementia. A second series of experiments was conducted on mice injected with six doses of aluminum hydroxide. Behavioural analyses in these mice revealed significant impairments in a number of motor functions as well as diminished spatial memory capacity. The demonstrated neurotoxicity of aluminum hydroxide and its relative ubiquity as an adjuvant suggest that greater scrutiny by the scientific community is warranted.

Source: J Inorg Biochem. 2009 Nov;103(11):1555-62. doi: 10.1016/j.jinorgbio.2009.05.019. Epub 2009 Aug 20

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2819810/

Radiologist Dr. David Ayoub Talks about Aluminium Toxicity

Clinical Immunologist Dr. Yehuda Shoenfeld Talks about Autoimmune Syndrome Induced by Adjuvants (ASIA)

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